By Kanu Chatterjee
Cardiac medicines describes the newest advances within the quickly evolving quarter of cardiovascular pharmacology. Written by means of well known, USbased specialists in cardiology, this publication discusses the drug teams for various illnesses, reminiscent of acute coronary syndromes, high blood pressure, angina and center failure. Separate chapters study cardiac medicines in being pregnant and lactation, and destiny instructions. more suitable through complete color photos and illustrations, this simple to stick to textual content deals sensible suggestion on tips on how to deal with cardiac ailments, with a spotlight on hands-on healing suggestions for clinicians.
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Extra resources for Cardiac Drugs
Efficacy and safety of the direct renin inhibitor aliskiren and ramipril alone or in combination in patients with diabetes and hypertension. J Renin Angiotensin Aldosterone Syst. 2007;8(4):190-8. 84. Andersen K, Weinberger MH, Egan B, Constance CM, Ali MA, Jin J, et al. Comparative efficacy and safety of aliskiren, an oral direct renin inhibitor, and ramipril in hypertension: a 6-month, randomized, double-blind trial. J Hypertens. 2008;26(3):589-99. 85. Oh BH, Mitchell J, Herron JR, Chung J, Khan M, Keefe DL.
2003;107(20):2555-8. 8. McDonald KM, Mock J, D’Aloia A, Parrish T, Hauer K, Francis G, et al. Bradykinin antagonism inhibits the antigrowth effect of converting enzyme inhibition in the dog myocardium after discrete transmural myocardial necrosis. Circulation. 1995;91(7):2043-8. 9. ONTARGET Investigators, Yusuf S, Teo KK, Pogue J, Dyal L, Copland I, et al. Telmisartan, ramipril, or both in patients at high risk for vascular events. N Engl J Med. 2008;358(15):1547-59. 10. Meredith PA, Ostergren J, Anand I, Puu M, Solomon SD, Michelson EL, et al.
It is currently approved for the treatment of hypertension in the USA and could potentially emerge as important therapy for HF. Aliskiren is a nonpeptide piperidine that inhibits the enzyme renin by binding to its catalytic site producing about 50% reduction in renin activity. Virtually, all the subsequent messengers for the RAAS receptors are then attenuated. This tends to offset the heightened renin activity when concomitant diuretics, ACEIs or ARBs are used. Whether this provides an important clinical advantage over ACEIs or ARBs alone has been controversial.