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Additional info for Cardiac arrhythmias 2005: proceedings of the 9th international workshop on cardiac arrhythmias - Venice, October 2-5 2005
Am J Med 113:365–370 Jaïs P, Shah DC, Haïssaguerre M et al (2000) Mapping and ablation of left atrial flutters. Circulation 101:2928–2934 Ouyang F, Ernst S, Vogtmann T et al (2002) Characterization of reentrant circuits in left atrial macroreentrant tachycardia: critical isthmus block can prevent atrial tachycardia recurrence. Circulation 105:1934–1942 Cosio FG, Martin-Penato A, Pastor A et al (2003) Atypical flutter: a review. Pacing Clin Electrophysiol 26:2157–2169 Olgin JE, Jayachandran JV, Engesstein E et al (1998) Atrial macroreentry involving the myocardium of the coronary sinus: a unique mechanism for atypical flutter.
Delise et al. ), which constitute the anatomic circuit of atrial flutter. Consequently, in many cases, as AF extinguishes, atrial flutter initiates. ’ Finally, the two arrhythmias may have a common trigger, represented by automatic foci of pulmonary veins . Antiarrhythmic drugs may have opposite effects on AF and atrial flutter. More precisely, many drugs (propafenone, flecainide, amiodarone) are able to prevent AF but they have little effect on, or may even facilitate atrial flutter. This observation has been made by several investigators, who demonstrated the occurrence of atrial flutter in 5–22% of patients with AF after treatment with class1 C drugs or amiodarone [10–14].
6 mV), but still viable area at the border zone of the scar tissue at a distance of 16 mm from the sinus node area. Radiofrequency energy delivery at the earliest activated site by cool-tip catheter (40 Watts) for 45 s produced early and sudden tachycardia termination, with no sign of damage to the sinus node. Two other applications, on sinus rhythm, were then delivered just superiorly and inferiorly to the tachycardia focus. Afterwards, the tachycardia was no longer inducible even at the maximal infusion rate of isoprenalin, and no other arrhythmia was observed.